Pathophysiological Framework
Long COVID involves complex changes in immune function, inflammatory signaling, and tissue integrity.
The persistence of viral components, autoimmunity, and mitochondrial dysfunction are key areas of investigation.
Degenerative and inflammatory processes disrupt the normal homeostatic balance, leading to multisystem symptoms including fatigue, cognitive impairment, and dysautonomia.
The regenerative approach we employ considers these pathophysiological aspects by addressing
multiple components of the post-viral cascade. Cellular therapy aims to modulate local and systemic
inflammatory environments while potentially supporting tissue repair. Concurrent rehabilitation strategies address the functional aspects of recovery, and nutritional support targets metabolic factors that may influence post-viral health.